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Extra resources for Advances in Immunology, Vol. 29
1980). P l H with respect to its interaction with factor B (see above). These observations suggest that a low sialic acid density on the surface of a particle is not required for alternative pathway activation and lend support to the “PlH-antagonist” hypothesis. It has been shown that an activator could be changed into a nonactivator by defined chemical modification. , 1979b). Approximately 12 million substitutions were required to render a zymosan particle a nonactivator, on whose surface C3b molecules are totally susceptible to the control proteins.
Shown areE. coli treated at 37°C for 60 minutes with buffer or the heat-inactivated (56"C,30 minutes) isolated component mixture (upper left panel), the isolated component mixture (upper right, lower left), or the isolated component mixture containing 10 pg of egg white lysozyme per milliliter (lower right). x 30,000. ALTERNATIVE PATHWAY OF COMPLEMENT 35 X . , 1980). , 1980). Certain cells are activators of the pathway by virtue of their expressing viral antigen on their surface. Other activating cells do not express detectable surface antigens of viral origin.
MULLER-EBERHARD AND ROBERT D. 1 Es, sheep erythrocyte; E H , human erythrocyte; BH-LPS, base-hydrolyzed lipopolysaccharide. , 1980). P l H with respect to its interaction with factor B (see above). These observations suggest that a low sialic acid density on the surface of a particle is not required for alternative pathway activation and lend support to the “PlH-antagonist” hypothesis. It has been shown that an activator could be changed into a nonactivator by defined chemical modification. , 1979b).